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INTRODUCTION Chest pain with concurrently elevated cardiac enzymes in a young patient with no known cardiovascular risk factors should prompt an exploration of the drug history. In recent years, anabolic steroids and weight loss supplements have started to gain a reputation for cardiac toxicity. Clenbuterol, a selective β 2-adrenergic receptor agonist, has been shown to induce cardiac hypertrophy in rats with molecular and functional parameters consistent with those of physiologic cardiac hypertrophy. The salutary effects of clenbuterol on the heart. IGF-I is known to exert many beneficial effects on the heart and can improve cardiac function in the failing heart in vivo,10 for example by attenuating the progression of heart failure in a model of dilated cardiomyopathy. Effects of clenbuterol on beta 1 and beta 2 adrenergic receptor mRNA levels in the heart of wild-type controls (n=4), MLP −/− treated with saline (n=4), and MLP −/− treated with clenbuterol (n=5). Expression levels were measured by RT-PCR and normalized to an HPRT1 transcript as an internal control. Clenbuterol (Clen), a β 2 -agonist, is known to produce skeletal and myocardial hypertrophy. This compound has recently been used in combination with left ventricular assist devices for the treatment of end-stage heart failure to reverse or prevent the adverse effects of unloading-induced myocardial atrophy. The β2-selective adrenoreceptor agonist clenbuterol promotes both skeletal and cardiac muscle hypertrophy and is undergoing clinical trials in the treatment of muscle wasting and heart failure From the classic bulking stack to the cutting stack, we’ve got you covered, clenbuterol heart hypertrophy.
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